NAD+ Explained
Unfortunately, as we age, our supply of NAD+ naturally disappears. By age 40, most people have lost about half of their youthful levels, and by age 60, they may have only 20% left. This happens because certain enzymes in the body start to "steal" and consume NAD+ much faster than we can replace it, usually to deal with rising inflammation or to try and fix mounting DNA damage. As NAD+ is used up by these "thieves," there is not enough left to run your power plants or fuel your repair crews. Begins Here
Metabolic Preservation Strategies: A Clinical Framework for Resistance Training and Nutrient Timing in GLP-1 Therapy
Emerging data indicate that 20% to 50% of total weight loss during GLP-1 RA therapy may consist of lean mass, with some cohorts showing muscle loss as high as 60%. This reduction in skeletal muscle integrity presents a significant risk for sarcopenic obesity, metabolic rate depression, and decreased functional independence.
The Hidden Risks of "Gray Market" Tirzepatide: Why Purity Matters for Your Long-Term Health
You may see Tirzepatide sold online or in local Albuquerque nutrition stores labeled as "for research use only" or "not for human consumption." While the lower price point is tempting, the long-term health consequences of using these unapproved substances can be severe and, in some cases, irreversible.
The Longevity Coenzyme: Clinical Efficacy of Subcutaneous Nicotinamide Adenine Dinucleotide in Addressing Age-Related Pathophysiology and Metabolic Decline
Subcutaneous (SC) injections have emerged as a superior modality for achieving therapeutic concentrations while mitigating the metabolic bottlenecks associated with oral ingestion and the systemic side effects inherent to rapid intravenous delivery. This analysis explores the mechanistic underpinnings of NAD+ therapy, the clinical superiority of the subcutaneous route, and the synergistic integration of NAD+ restoration with emerging medical technologies and supportive measures in managing the hallmarks of aging.